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NEW QUESTION: 1

A. Access Team
B. User or Team
C. Organization
D. Owner
Answer: B

NEW QUESTION: 2
From a compliance and security perspective, which of these statements is true?
A. You do not ever need to rotate access keys for AWS IAM Users.
B. You do not ever need to rotate access keys for AWS IAM Roles, nor AWS IAM Users.
C. You do not ever need to rotate access keys for AWS IAM Roles.
D. None of the other statements are true.
Answer: C
Explanation:
IAM Role Access Keys are auto-rotated by AWS on your behalf; you do not need to rotate them.
The application is granted the permissions for the actions and resources that you've defined for the role
through the security credentials associated with the role. These security credentials are temporary and we
rotate them automatically. We make new credentials available at least five minutes prior to the expiration
of the old credentials.
Reference: http://docs.aws.amazon.com/AWSEC2/latest/UserGuide/iam-roles-for-amazon-ec2.html

NEW QUESTION: 3
You administer a Microsoft Azure SQL Database data base in the US Central region named contosodb. Contosodb runs on a Standard tier within the S1 performance level.
You have multiple business-critical applications that use contosodb.
You need to ensure that you can bring contosodb back online in the event of a natural disaster in the US Central region. You want to achieve this goal with the least amount of downtime.
Which two actions should you perform? Each correct answer presents part of the solution.
A. Use point in time restore.
B. Upgrade to Premium tier.
C. Downgrade to Basic tier.
D. Use active geo-replication.
E. Use automated Export.
F. Upgrade to S2 performance level.
Answer: B,D

NEW QUESTION: 4
Select the class of Anti-diabetic medication that works in the specified organ to prevent hyperglycemia. Select all that applies. GI tract (B)
A. Sulfonylureas
B. Biguanide
C. Thiazolidinediones
D. Alpha- Glucosidase Inhibitors
E. SGLT2 inhibitors
F. DPP4 Inhibitors
G. Glucagon-like peptide-1 receptor agonists
Answer: G
Explanation:
Explanation
Sulfonylureas work in beta cells in the pancreas that are still functioning to enhance insulin secretion. Alpha- Glucosidase Inhibitors stop -glucosidase enzymes in the small intestine and delay digestion and absorption of starch and disaccharides which lowers the levels of glucose after meals. DPP4 blocks the degradation ofGLP-1, GIP, and a variety of other peptides, including brain natriuretic peptide. Glucagon-like peptide-1 receptor agonists work in various organs of the body. Glucagon-like peptide-1 receptor agonists enhance glucose homeostasis through: (i) stimulation of insulin secretion; (ii) inhibition of glucagon secretion; (iii) direct and indirect suppression of endogenous glucose production; (iv) suppression of appetite; (v) enhanced insulin sensitivity secondary to weight loss; (vi) delayed gastric emptying, resulting in decreased postprandial hyperglycaemia.Thiazolidinediones are the only true insulin-sensitising agents, exerting their effects in skeletaland cardiac muscle, liver,and adipose tissue. It ameliorates insulin resistance, decreases visceral fat.Biguanides work in liver, muscle, adipose tissue via activation of AMP-activated protein kinase (AMPK) reduce hepatic glucose production. SGLT2 inhibitors work in the kidneys to inhibit sodium-glucose transport proteins to reabsorb glucose into the blood from muscle cells; overall this helps to improve insulin release from the beta cells of the pancreas.